14. The Role of Copper and Zinc Toxicity in Innate Immune Defense against Bacterial Pathogens, https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4521016/
15. Copper and immunity, https://pubmed.ncbi.nlm.nih.gov/9587153/
16. Partners in crime: neutrophils and monocytes/macrophages in inflammation and disease, https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5820413/
17. Copper regulates cyclic-AMP-dependent lipolysis, https://pubmed.ncbi.nlm.nih.gov/27272565
18. Human copper transporters: mechanism, role in human diseases and therapeutic potential, https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2863355/
19. Using Copper to Improve the Well-Being of the Skin, https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4556990/
20. Effects of antioxidant supplementation on the aging process, https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2685276/
21. Reduced zinc and copper in the retinal pigment epithelium and choroid in age-related macular degeneration, https://pubmed.ncbi.nlm.nih.gov/18848316/
22. Copper, magnesium, zinc and calcium status in osteopenic and osteoporotic post-menopausal women, https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4469220/
23. Denture cream: an unusual source of excess zinc, leading to hypocupremia and neurologic disease, https://pubmed.ncbi.nlm.nih.gov/18525032/
24. Influence of ascorbic acid supplementation on copper status in young adult men, https://pubmed.ncbi.nlm.nih.gov/6837490/
25. Copper deficiency anemia: review article, https://pubmed.ncbi.nlm.nih.gov/29959467/
26. Hepatic iron overload or cirrhosis may occur in acquired copper deficiency and is likely mediated by hypoceruloplasminemia, https://pubmed.ncbi.nlm.nih.gov/20502350/
27. Copper homeostasis at the host-pathogen interface, https://pubmed.ncbi.nlm.nih.gov/22389498/
28. Copper excess, zinc deficiency, and cognition loss in Alzheimer's disease, https://pubmed.ncbi.nlm.nih.gov/22438177/
29. Copper deficiency alters cell bioenergetics and induces mitochondrial fusion through up-regulation of MFN2 and OPA1 in erythropoietic cells, https://pubmed.ncbi.nlm.nih.gov/23831624/
30. Copper deficiency presenting as metabolic bone disease in extremely low birth weight, short-gut infants, https://pubmed.ncbi.nlm.nih.gov/22869833/
31. Inherited copper transport disorders: biochemical mechanisms, diagnosis, and treatment, https://pubmed.ncbi.nlm.nih.gov/21838703/
Молибден (Mo)
Молибден – полезный минерал, о котором вы вряд ли слышали. Он нужен организму в крошечных количествах, зато этого достаточно для участия во многих жизненно важных процессах. Без него в теле накапливаются токсины, не усваиваются аминокислоты, не обрабатывается ДНК. В этой статье разбираемся, почему он так важен для здоровья и рассказываем все, что вам нужно знать о малоизвестном микроэлементе.